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It must be reminded that in our experience, IGF-1 treatment does not only raise IGF-1 plasma levels, but also stimulates tissues to synthesise their own IGF-1 by a mechanism still not uncovered, aiming these mechanisms of central stimulation of peripheral improvement

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This is probably, due to diverse mechanisms such as differences in the thiol content, NTR-I downregulation (Wilkinson et al., 2008), or deletion of copies of the gene encoding prostaglandin F2 synthase (referred as old yellow enzyme) that is involved in activating the anti-parasite drugs (Murta et al., 2006)

The dynamic abilities of glutathione are indefinite