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Fig ure 2 Microscopic presentation of intestinal adaptation in diclofenac-treated animals

These modifications are designed to counteract the immunosuppressive tumor microenvironment (TME) by enhancing T-cell activity and survival, thereby directing CAR-T cells towards the tumor site

Compared with the rats treated with APAP alone, rhein treated animals demonstrated significant reduction of the biochemical indicators of liver injury, including aspartate aminotransferase (AST), alanine aminotransferase (ALT), urea nitrogen (UREA), creatinine (CREA), nitric oxide (NO), and malondialdehyde (MDA)