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Two major Ca 2+ entry pathways are important for NO production: store-operated Ca 2+ entry (SOCE), the main agonist-induced Ca 2+ entry mechanism in ECs (Tran, 2020), and mechanosensitive Ca 2+ entry, stimulated by blood shear stress

Among the mechanisms that determine disease severity, immature molecular response and defense systems at the intestinal epithelial cell (IEC) are implicated

Adverse Effects Common: somnolence, anorexia, small increase in blood pressure and pulse

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