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Based on this, in the future, encapsulating GLP-1 analogs within vitamin A-modified liposomes, while simultaneously targeting HSCs and leveraging the anti-inflammatory and metabolic regulatory effects of GLP-1 analogs, is expected to synergistically reduce HSC activation and improve the hepatic metabolic environment, thus offering a more promising innovative therapy for NASH and related liver fibrosis [254, 255]

None of these represent a proven cancer-prevention or cancer-treatment effect they're plausible biological pathways based on what's already understood about metabolism, inflammation, and tumor growth in general

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2025 Dec;85(12):1621-1627