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Description

As the central hub for iron metabolism and ROS production, mitochondria not only initiate ferroptotic signaling but also serve as promising therapeutic targets

The approach remains theoretical but aligns with general principles of supporting recovery during periods of elevated repair activity

Avoid after 34pm due to stimulant strength

Treatment with RSL3 (a ferroptosis inducer) has been found to downregulate SLC2A3 expression and induce ferroptosis in RA fibroblast-like synoviocytes (RA-FLS) [163]