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Sean focuses on operations, IT, and the financials for the group

This activation enhances glutamate release from primary afferent terminals, resulting in increased normal and miniature excitatory postsynaptic currents, and elevates PKC activity, which mediates the phosphorylation of NMDARs (e.g., the GluN1 subunit at Ser896)

Conflict of interest The authors have no conflicts of interest regarding the research described in this paper

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