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Engaging all three pathways simultaneously drives glucose-dependent insulin secretion through the two incretin arms while adding glucagon-receptor agonism implicated in energy expenditure, the pharmacological rationale that distinguishes it from single (GLP-1 (Sema)) and dual (GLP-2 (Triz)) incretin agonists

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The bottom line: peptide protocols for autoimmune conditions should never be designed or implemented without the involvement of a qualified healthcare provider who understands both the conventional treatment regimen and the peptide mechanisms involved

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