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In neoplastic cells, activation of NF-B provides the ability to survive by upregulating anti-apoptotic genes, including several members of the BCL-2 family [192], and increases resistance to chemotherapy by controlling the expression of multidrug resistance gene 1 (mdr1) [193]
Personalized medicine strategies, targeting individuals' specific biological pathways affected in ASD, could position sulforaphane as a versatile and supportive option
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