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Funding Statement The authors are supported by grants from the National Institute of Health (NIH/NCCIH)-(5R33AT010637-01A1 and 3R33 AT010637-02S1) and the Malcolm Feist Endowed Chair in Diabetes

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We also evaluated the ability of conditioned media (CM) from pre-treated PCa and BPH cells with ALCAR (1 or 10 mM), to interfere with the expression of CXCR4 (receptor for CXCL12), CXCR1, CXCR2 (receptors for CXCL8) and CCR2 (receptor for CCL2) on human umbilical vein endothelial cells (HUVEC