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NASH: nonalcoholic steatohepatitis

3.7.3 Mechanisms of association between UPF intake and ASD There have been a range of recent studies that have shown that high dietary intake of UPF may contribute to an increased risk of developing ASD, which may be mediated through a range of different pathophysiological mechanisms, including lipid dysregulation, oxidative stress and neuroinflammation (Table 2)

These medicines act in a similar way to a natural hormone called glucagon-like peptide-1, which helps regulate blood sugar by prompting the pancreas to release insulin when glucose levels rise, and by slowing how quickly the stomach empties

Follow the instructions carefully regarding dosage and frequency of application