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Primary cells from CLL patients were unresponsive to CB-839mediated apoptosis induction and mitochondrial respiration inhibition

doi: 10.1042/bsr20230374 35 MartinPRSingletonCKHiller-SturmhfelS

However, a functional role of mitochondrial disease in ASD phenotype has been proposed by some researchers [74]

The first runs through phosphoinositide 3-kinase (PI3K), Akt (protein kinase B), and mechanistic target of rapamycin complex 1 (mTORC1), which phosphorylates p70-S6 kinase and 4E-BP1 to accelerate ribosomal protein translation [3]