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Second, carotenoid-dependent mechanism, or quenching , is predicated on the power of PS II to transfer electrons to varied acceptors in its proximity (carotenoids, -tocopherol), which liberate excess energy within the sort of heat, returning to their ground state (Trebst, 2003

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In PTSD mice models, trauma exposure increased tyrosine hydroxylase expression (the rate-limiting enzyme in catecholamine biosynthesis), elevating stress hormones epinephrine and norepinephrine in serum and colon, which upregulated claudin-2 expression, confirming gut barrier dysfunction (Yadav et al., 2023)