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1 To date, multiple mechanisms of tissue damage have been implicated in SLE, including oxidative stress and free radicals, fibrosis, complement-mediated pathways, and a panoply of enzymatic cascades including matrix metalloproteinases, the renin-angiotensin system, plasmin, and kallikreins

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Tumor necrosis factor alpha mediates resistance to Trypanosoma cruzi infection in mice by inducing nitric oxide production in infected gamma interferon-activated macrophages