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Overexpression of TB-500 in cells appears to cause an increased growth rate, reduced basal apoptosis, and resistance to cell death-inducing factors

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It pairs an N-acetylated Semax backbone with a C-terminal adamantane modification (borrowed from Peptide 021/P21), engineered for enhanced metabolic stability, blood-brain-barrier penetration, and extended duration of BDNF-modulating activity