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In activated T cells, flux through the oxidative PPP increases nearly 10-fold, but chemical inhibition of G6PD can markedly decrease this and, as a result, impair T cell activation and decrease production of pro-inflammatory interferon-gamma (IFN-) (55)

Nardin, A., Lefebvre, M

In contrast, induction of ER stress via central administration of thapsigargin completely abolished the RYGB-mediated improvement in leptin sensitivity, without affecting control animals, highlighting ER stress as a key regulatory factor in the beneficial effects of RYGB on hypothalamic leptin signaling [97]