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increased Desulfovibrionaceae ) promotes plaque formation through metabolic inflammation ( Caspase1 -/- mouse microbiota demonstrate accelerated atherosclerosis in Ldlr -/- recipients through systemic inflammation and microbial community shifts ( 3.3 TMAO as a promoter of atherosclerosis TMAO accelerates atherosclerosis via multiple mechanisms

In particular, hypoxia was unique to CD8 + T cells and the G2-M checkpoint was shared by CD4 + T and plasma cells in the Hallmark gene set ( Figure 8I )

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