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doi: 10.1152/jappl.2001.91.2.693

doi: 10.1021/bm500899r

Whether these are important mechanisms of GSH signaling under metal stress needs yet to be revealed

Thus, damage to mitochondrial DNA and loss of can further increase ROS production, thereby creating a cycle of dysfunction that includes increased ROS production by the respiratory chain, activation of signal transduction pathways, and potentially, leading to apoptosis or necrosis, as well as the release of cytochrome c, which triggers apoptosis