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Neurological Relevance Preclinical evidence suggests broader neuroprotective potential: In traumatic brain injury models, SS-31 reduced mitochondrial swelling, neuronal loss, and neuroinflammation Aged mice treated with SS-31 showed significantly improved exercise tolerance, muscle strength, and mitochondrial efficiency Studies demonstrate protection against ischemic injury and enhanced cardiac output Phase 3 trials are ongoing for dry age-related macular degeneration and primary mitochondrial myopathy (Stealth BioTherapeutics pipeline) Implications for Brain Aging Mitochondrial dysfunction is a hallmark of brain aging and neurodegeneration

GHK- Cu , also known as the copper peptide, is renowned for its potential involvement in tissue regeneration, collagen synthesis, wound healing, and skin regeneration

PDRN also inhibits microphthalmia-associated transcription factor (MITF), which reduces melanin synthesis

Refer to the batch-specific COA